The Saved Joe Donnelly’s Knee Stack

Research Protocol for the Saved Joe Donnelly’s Knee Stack:

BPC-157 – Prior to research, BPC-157 should first be reconstituted with bacteriostatic water by injecting bacteriostatic water in the vial of BPC-157. A 500MCG dose (1/10th of the bottle) should be used every day. BPC-157 can be researched until healing is complete or after 3 months of research. Following 3-months of usage, a 1-month break should be taken before resuming research.

TB-500 – Prior to research, TB-500 should first be reconstituted with bacteriostatic water by injecting bacteriostatic water in the vial of TB-500. A 500MCG dose (1/20th of the bottle) should be used twice a week. TB-500 can be researched until healing is complete, or for up to 6 weeks. Following 6 weeks of usage, a 2 week break should be taken before resuming research.

BPC-157

BPC-157 (also known as PL 14736) is a pentadecapeptide. It has the amino acid sequence Gly-Glu-Pro-Pro-Pro-Gly-Lys-Pro-Ala-Asp-Asp-Ala-Gly-Leu-Val. This peptide has a molecular formula of C62H98N16O22. The PUBCHEM ID is CID 9941957.

There are some tentative pre-clinical studies on animals and in vitro suggesting possible benefit in wound healing and bowel disorders although all of these studies come from a single research group.

TB-500

Work with cell cultures and experiments with animals have shown that administration of thymosin β4 can promote migration of cells, formation of blood vessels, maturation of stem cells, survival of various cell types and lowering of the production of pro-inflammatory cytokines. These multiple properties have provided the impetus for a worldwide series of on-going clinical trials of potential effectiveness of thymosin β4 in promoting repair of wounds in skin, cornea and heart.

Such tissue-regenerating properties of thymosin β4 may ultimately contribute to repair of human heart muscle damaged by heart disease and heart attack. In mice, administration of thymosin β4 has been shown to stimulate formation of new heart muscle cells from otherwise inactive precursor cells present in the outer lining of adult hearts, to induce migration of these cells into heart muscle and recruit new blood vessels within the muscle.

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